来自四川大学的Liu等 (2015) 报道了一个糖基转移酶基因 UGT71C5 能够催化拟南芥ABA的糖基化，突变株或下调 UGT71C5 表达能延迟种子萌发及增强抗旱能力，此时ABA的含量升高；而 UGT71C5 过表达时则情况相反。作者建议UGT71C5将ABA糖基化，便于从细胞质中运输到液泡中储存；在逆境条件下，通过AtBG1/2基因去除糖基又释放到细胞质中，增加游离ABA的含量，行使逆境生理功能。 Liu, Z., Yan, J. P., Li, D.K., Luo, Q., Yan, Q., Liu, Z. B., Ye, L. M., Wang, J. M., Li, X. F., Yang, Y.,2015. UDP-Glucosyltransferase71C5, a Major Glucosyltransferase, MediatesAbscisic Acid Homeostasis in Arabidopsis. Plant Physiology 167, 1659-1670.
你要的论文The ABA Receptor PYL8 Promotes Lateral Root Growth by Enhancing MYB77-Dependent Transcription of Auxin-Responsive Genes已经下载到了，请下载本文附件。 Abstract The phytohormone abscisic acid ( ABA ) regulates plant growth, development , and abiotic stress responses. ABA signaling is mediated by a group of receptors known as the PYR1 /PYL/ RCAR family, which includes the pyrabactin resistance 1-like protein PYL8. Under stress conditions, ABA signaling activates SnRK2 protein kinases to inhibit lateral root growth after emergence from the primary root. However, even in the case of persistent stress, lateral root growth eventually recovers from inhibition. We showed that PYL8 is required for the recovery of lateral root growth, following inhibition by ABA . PYL8 directly interacted with the transcription factors MYB77, MYB44, and MYB73. The interaction of PYL8 and MYB77 increased the binding of MYB77 to its target MBSI motif in the promoters of multiple auxin -responsive genes. Compared to wild-type seedlings, the lateral root growth of pyl8 mutant seedlings and myb77 mutant seedlings was more sensitive to inhibition by ABA . The recovery of lateral root growth was delayed in pyl8 mutant seedlings in the presence of ABA , and the defect was rescued by exposing pyl8 mutant seedlings to the auxin IAA (3-indoleacetic acid). Thus, PYL8 promotes lateral root growth independently of the core ABA -SnRK2 signaling pathway by enhancing the activities of MYB77 and its paralogs, MYB44 and MYB73, to augment auxin signaling. The ABA Receptor PYL8 Promotes Lateral Root Growth by.pdf
标签: ABA
